Circulating low-grade inflammation in human pulmonary arterial hypertension promotes eNOS-NO/ROS imbalance: Involvement of the angiotensin and the endothelin pathways and prevention by EPA:DHA 6:1
Inflamed atrial tissues of patients with atrial fibrillation are characterized by pro-oxidant and pro-remodeling responses: synergistic inhibition by an SGLT2 inhibitor plus a GLP-1R activation
Reversal of age-related diastolic dysfunction by micronized purified flavonoid fraction in rats with attenuation of low-grade inflammation, eNOS-NO/ROS imbalance, and left ventricular fibrosis
Speaker:
Professor V. Schini-Kerth (Strasbourg, FR)