ESC 365 - Concomitant genetic ablation of L-type Cav1.3 and T-type Cav3.1 Ca2+ channels disrupts heart automaticity.

EHRA Premium Access

Concomitant genetic ablation of L-type Cav1.3 and T-type Cav3.1 Ca2+ channels disrupts heart automaticity.

Session: (Sino-)Atrial electrophysiology and cardiac arrhythmia

Topic: Ion Channels, Electrophysiology

Speaker: Doctor M. Mangoni (Montpellier, FR)

Event: EHRA 2019

Congress Presentation

Add to my library

About the speaker

Doctor Matteo Elia Mangoni

Institute of Functional Genomics, University of Montpellier, Montpellier (France)

2 presentations

0 follower

4 more presentations in this session

Intracellular calcium handling in different models of sinus node dysfunction

Speaker: Doctor A. Gomez (Orsay, FR)

Massive expansion of native human atrial cardiomyocytes by immortogenetics for multiscale arrhythmia research

Speaker: Mr N. Harlaar (Leiden, NL)

Investigation of the Na+/Ca2+-exchanger-funny current crosstalk in sinus-node and Purkinje fibre assessed by a novel NCX inhibitor ORM-10962

Speaker: Doctor N. Toth (Szeged, HU)

Gain-of-function mutation in the cardiac kv4.2 potassium channel underlies paroxysmal atrial fibrillation

Speaker: Mr M. Drabkin (Beer Sheva, IL)

Access the full session

(Sino-)Atrial electrophysiology and cardiac arrhythmia

Speakers: Doctor M. Mangoni, Doctor A. Gomez, Mr N. Harlaar, Doctor N. Toth, Mr M. Drabkin

About the event

EHRA 2019

17 March - 19 March 2019

Sessions Presentations

Congress

ESC 365 is supported by

Explore sponsored resources

Explore sponsored resources