Doctor M Rabiul Hosen

Doctor M Rabiul Hosen

University hospital Bonn, Bonn (Germany)

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Dr. Rabiul Hosen is a Senior Postdoctoral Researcher in Molecular Cardiology, Heart Centre Bonn, University of Bonn, Germany. After obtaining a BS in Genetic Engineering and Biotechnology from Bangladesh, he traveled to Germany MS in Biomedical Science from Bonn and Molecular Stem Cell Biology from Bochum, and a Ph.D. from University Frankfurt where he studied noncoding RNAs. His concurrent research consists of the diverse analysis of noncoding genomes, especially, long noncoding RNA (lncRNA) which has enormous perspective in different pathways. Dr. Hosen exclusively focusing on cardiovascular disease (CVD) to study the underlying molecular mechanisms of pathogenesis in coronary heart diseases (CHDs) and the development of holistic mRNA- and noncoding RNA-based therapeutic approaches for the treatment of multifactorial CVDs. He also working on the development of RNA-based cardiovascular biomarkers via “Liquid Biopsy” and therapies by focusing on different cardiovascular diseases.

Presentations

Extracellular vesicle-incorporated long noncoding RNA PUNISHER is increased in coronary artery disease and regulates endothelial cell function

Event: ESC Congress 2024

Topic: Pathophysiology and Mechanisms

Session: Advancements in cardiovascular medicine: from molecular insights to clinical strategies in heart disease management

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Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response and endothelial cells function via NFkB-dependent mechanism

Event: ESC Congress 2024

Topic: Pathophysiology and Mechanisms

Session: Experimental studies in coronary atherosclerosis disease

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Aortic valve disease augments vesicular microRNA-145-5p to regulate the calcification of valvular interstitial cells via cellular crosstalk

Event: ESC Congress 2024

Topic: Pathophysiology and Mechanisms

Session: New molecular insights into valvular heart disease

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Calcific aortic valve disease-associated long noncoding RNA H19 promotes the osteogenic transition of valvular interstitial cells via the JAG1/NOTCH1 axis

Event: ESC Congress 2024

Topic: Pathophysiology and Mechanisms

Session: New molecular insights into valvular heart disease

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Aortic valve stenosis augments long noncoding RNA H19 to govern angiogenic responses and phenotypes of valvular endothelial cell through endothelial to mesenchymal transition

Event: ESC Congress 2024

Topic: Aortic Valve Stenosis

Session: New molecular insights into valvular heart disease

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Long noncoding RNA H19 is elevated in aortic valve stenosis and governs antigenic response and valvular endothelial cell phenotypes through endothelial to mesenchymal transition

Event: ESC Congress 2023

Topic: Aortic Valve Stenosis

Session: From bedside to bench: unravelling the mechanisms of valvular heart disease

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Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response and endothelial cells function via NFkB-dependent mechanism

Event: ESC Congress 2022

Topic: Biomarkers

Session: Following disease progression by blood biomarkers

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Circulating microRNA-122-5p is associated with a lack of improvement in left-ventricular function after TAVR and regulates viability of cardiomyocytes via extracellular vesicles

Event: ESC Congress 2022

Topic: Ventricular Remodelling

Session: Chronic heart failure - Ventricular

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Circulating microRNA-122-5p correlates with improvement in left-ventricular function after transcatheter aortic valve replacement and regulates viability of cardiomyocytes via extracellular vesicles

Event: ESC Congress 2021 - The Digital Experience

Topic: Microvesicles, Exosomes

Session: Young Investigator Awards Session in Basic Science

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Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response andendothelial cells function via NFkB-dependent mechanism

Event: ESC Congress 2021 - The Digital Experience

Topic: Biomarkers

Session: Acute Coronary Syndromes ePosters

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Doctor M Rabiul Hosen

Biography

Contributor content

Extracellular vesicle-incorporated long noncoding RNA PUNISHER is increased in coronary artery disease and regulates endothelial cell function

Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response and endothelial cells function via NFkB-dependent mechanism

Aortic valve disease augments vesicular microRNA-145-5p to regulate the calcification of valvular interstitial cells via cellular crosstalk

Calcific aortic valve disease-associated long noncoding RNA H19 promotes the osteogenic transition of valvular interstitial cells via the JAG1/NOTCH1 axis

Aortic valve stenosis augments long noncoding RNA H19 to govern angiogenic responses and phenotypes of valvular endothelial cell through endothelial to mesenchymal transition

Long noncoding RNA H19 is elevated in aortic valve stenosis and governs antigenic response and valvular endothelial cell phenotypes through endothelial to mesenchymal transition

Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response and endothelial cells function via NFkB-dependent mechanism

Circulating microRNA-122-5p is associated with a lack of improvement in left-ventricular function after TAVR and regulates viability of cardiomyocytes via extracellular vesicles

Circulating microRNA-122-5p correlates with improvement in left-ventricular function after transcatheter aortic valve replacement and regulates viability of cardiomyocytes via extracellular vesicles

Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response andendothelial cells function via NFkB-dependent mechanism

ESC 365 is supported by

Doctor M Rabiul Hosen Follow

Biography

Contributor content

Extracellular vesicle-incorporated long noncoding RNA PUNISHER is increased in coronary artery disease and regulates endothelial cell function

Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response and endothelial cells function via NFkB-dependent mechanism

Aortic valve disease augments vesicular microRNA-145-5p to regulate the calcification of valvular interstitial cells via cellular crosstalk

Calcific aortic valve disease-associated long noncoding RNA H19 promotes the osteogenic transition of valvular interstitial cells via the JAG1/NOTCH1 axis

Aortic valve stenosis augments long noncoding RNA H19 to govern angiogenic responses and phenotypes of valvular endothelial cell through endothelial to mesenchymal transition

Long noncoding RNA H19 is elevated in aortic valve stenosis and governs antigenic response and valvular endothelial cell phenotypes through endothelial to mesenchymal transition

Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response and endothelial cells function via NFkB-dependent mechanism

Circulating microRNA-122-5p is associated with a lack of improvement in left-ventricular function after TAVR and regulates viability of cardiomyocytes via extracellular vesicles

Circulating microRNA-122-5p correlates with improvement in left-ventricular function after transcatheter aortic valve replacement and regulates viability of cardiomyocytes via extracellular vesicles

Coronary artery disease ameliorates extracellular vesicle lncRNA PUNISHER regulates angiogenic response andendothelial cells function via NFkB-dependent mechanism

ESC 365 is supported by

Doctor M Rabiul Hosen